renal cirrhosis

renal cirrhosis的基本解释

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[医] 肾硬变

与renal cirrhosis相关的网络例句 [注：此内容来源于网络，仅供参考]

White nails, found in the accident and loss of blood and shock; chronic diseases are found in anemia, hookworm disease, chronic gastrointestinal bleeding, tuberculosis hemoptysis, pulmonary heart disease and so on; as if a nail white glass, while the characteristics of liver cirrhosis; nail change White, thin, soft, was particularly prevalent in the chronic wasting disease; yellow nail is the lack of vitamin E hypothyroidism, nephrotic syndrome, and other symbolic; gray nails are often suffering from Onychomycosis, a performance for the initial stage next to the itching, Following the nail is deformed, but was dull gray; purple nails with a small red thorn is caused by hypoxia, indicating pulmonary heart disease; nail half red, half white (commonly known as a yin and yang) is not a manifestation of renal function; white nail Flocculation, or white, zinc deficiency may be, anemia or gastrointestinal system is sick; nail spots is the emergence of the performance of the poisoning; redness around the nail, mostly lupus erythematosus and in patients with dermatomyositis; nail root A blue and a half months is a reflection of peripheral circulation; obvious red nails, is the performance of heart failure; a clear and a half months, prompted the digestive system function better absorption; On the other hand, there is no clearance on a narrow or a half months, that is, Tip of the digestive system problems.

指甲变白时，急症见于失血、休克；慢性病则见于贫血、钩虫病、消化道慢性出血、肺结核咯血、肺源性心脏病等；如果指甲白得像毛玻璃一样，则为肝硬化的特征；指甲变白、变薄、变软，多见于慢性消耗疾病；指甲变黄是缺乏维生素E甲状腺功能减退、肾病综合症等象征；指甲变灰往往是患了甲癣，表现为初期甲旁发痒，继则指甲变形，失去光泽而呈灰白色；指甲青紫伴有红色小刺是缺氧引起的，预示肺心病；指甲一半红色、一半白色是肾功能不好的体现；指甲出现白点或絮状白斑，可能是缺锌、贫血或胃肠道系统有病；指甲出现黑斑是中毒的表现；指甲周围出现红斑，多见红斑性狼疮和皮肌炎患者；指甲根部的甲半月呈蓝色是末梢循环不良的反映；指甲明显发红，是心力衰竭的表现；甲半月明显者，提示消化系统吸收功能较好；反之，没有甲关月或甲半月窄小，即提示消化系统有毛病。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后，血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少，Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少，血小板聚集程度及红细胞聚集指数显著上升，病理切片显示胸主动脉内皮细胞破坏明显，脑心肾组织内的血管进一步增厚甚至几乎阻塞，导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR，脑体重比显著升高；心肌内出现明显的缺血梗塞疤痕灶，心体重比显著升高；肾小球明显萎缩、玻璃样变性，肾小管也变性甚至坏死，使肾硬化程度显著增加呈颗粒性固缩肾，肾体重比也显著减少。

Immunosuppression after renal transplantation seems to enhance viral replication and increase the risk of developing cirrhosis and hepatocellular carcinoma.

肾移植后的免疫抑制疗法似乎可以促进病毒复制，增加患者发生肝硬化和肝癌的风险。