regulates

regulates的基本解释

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[变形] regulate的第三人称单数

vt.

调节, 控制, 调整, 校准, 管理

与regulates相关的网络例句 [注：此内容来源于网络，仅供参考]

One set projects to the basal forebrain and regulates EEG components of REM sleep, whereas the other projects to the medulla and spinal cord and regulates atonia during REM sleep.

一方面刺激基底前脑并调控快动眼睡眠的脑电波，另一方面刺激髓鞘和脊髓使其在快动眼睡眠时处于松弛状态。

The bodies have much greater potential to regulate spermatogonial stem cell proliferation and differentiation. The dosage of GFRα1 mRNA produced by undifferential spermatogonial cells regulates the signal transduction of glial cell-derived neurotrophic factor so as to regulates the direction of spermato gonial stem cells: at a low level, spermatogonoial stem cell favors differentiation; at a high level, spermatogonial stem cell favors self-proliferation.

GFRαl在小鼠生精恢复过程中以剂量相关性调控著胶质细胞源性神经营养因数资讯传人，从而调控著精原干细胞的去向：高剂量时诱导其增殖，低剂量时诱导其分化。

Results are as followed:1 Exposure of HELF cells to BP caused c-Jun activation,and increased the activity of MAPK,PI-3K,p53 and cyclin D1 pathway.2 BP-induced c-Jun activation was inhibited by dominant negative mutants of extracellular signal-regulated protein kinase or c-Jun NH_2-terminal kinase,but not by p38,impling that JNK and ERK pathways medicate c-Jun activation induced by BP.3 Overexpression of dominant-negative mutants PI-3K and Akt potently blocked phosphorylations of c-Jun and ERK,but not JNK in response to BP,suggesting that PI-3K/Akt pathway positively regulates BP-induced c-Jun activation through ERK.4 Inhibition of p53 by its chemical or molecular inhibitor markedly increased the phosphorylation levels of c-Jun,Akt and ERK upon BP stimulation,indicating that p53 negatively medicates BP-induced c-Jun activation through PI-3K/Akt/ERK pathway.5 The cell lines expressed TAM67 exhibits no significant affecting normal cell growth properties.6 TAM67 was able to significantly block G_1-S transition and subsequent cell proliferation,suggesting that c-Jun is essential for cell cycle alternations elicited by BP.7 Overexpression of TAM67 impaired BP-induced cyclin D1 activation,decreasing expression of E2F1 and pRb,indicating that c-Jun participates in the modulation of BP-induced activation of cyclin D1/pRb/E2F1 pathway.8 Stably expression of TAM67 led to the increases in the expression levels of p53 and p21,elevating phosphorylation level of p53,clearly indicating that c-Jun regulates p53/p21 pathway activation induced by BRCollectively,PI3K/Akt/ERK pathway mediated BP-induced c-Jun activation through p53-dependent mechanism.

结果显示：1BP刺激细胞可促进c-Jun活化，并伴随着MAPK、PI-3K、p53和cyclinD1通路各组成成分的活性增强。2利用MAPK通路的显性失活突变体分别阻断细胞外信号调节激酶和c-Jun氨基末端激酶活性，均可明显抑制BP诱导的c-Jun活化，但阻断p38活性对BP引起的c-Jun活化无明显影响，提示JNK和ERK通路参与调控BP诱导的c-Jun活化。3过表达PI-3K和Akt的显性失活突变体也可显著抑制BP诱导的c-Jun活化，并降低磷酸化ERK的表达水平，但对磷酸化JNK的表达水平无明显影响，说明PI-3K/Akt通路通过ERK正性调控了BP诱导的c-Jun活化。4p53的化学/分子抑制剂能使BP作用的细胞内c-Jun活性明显增加，并同时诱导Akt和ERK的磷酸化水平的升高，表明p53可通过PI-3K/Akt/ERK通路对BP诱导的c-Jun活化进行负性调控。5随后观察转染细胞的生长情况，发现TAM67对细胞正常生长和形态无明显影响。6稳定表达TAM67可有效抑制BP诱导的S期细胞数的增加，提示c-Jun在BP致细胞周期改变的过程中发挥了重要作用。7TAM67过表达能够抑制BP诱导的cyclin D1活化，降低磷酸化Rb以及E2F1蛋白表达水平，表明c-Jun参与调控BP诱导的cyclin D1/Rb/E2F1通路的活化。8过表达TAM67可使BP刺激的细胞中p53、p21总蛋白以及p53磷酸化的表达水平明显升高，可见c-Jun也参与调控BP诱导的p53/p21通路活化。

与regulates相关的网络解释 [注：此内容来源于网络，仅供参考]

Regulates Qi：调气

Regulates Qi调气; | boosts Yang补阳; | supplements Qi补气;

Regulates body weight：调控体重

Iodine碘 | Regulates body weight调控体重 | Iron铁

sowing width regulates：播幅调整

sowing width number ==> 播幅大小 | sowing width regulates ==> 播幅调整 | sown area ==> 播种面积

Regulates bowel movements：调节肠运动

Rids the body of excess waste肋身体多余的废物 | Regulates bowel movements调节肠运动 | It's Free!它是免费的!

regulates trade in wildlife：管制野生动物买卖

监察水质monitor water quality | 管制野生动物买卖regulates trade in wildlife | 绿化工作greening work