arterially
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Using an LQT2 model in arterially perfnsed rabbit left ventricular wedge preparations,we find that in intact ventricular walI,EAD can be generated and a trigger to initiate the onset of Tdp can be produced.
结论通过冠状小动脉灌注兔左室心肌组织块LQT2模型,发现整体心室肌组织在QT延长的条件下,2相EAD是触发并引起Tdp的机制;TDR增加是产生EAD和形成折返的基础。
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Furthermore, we analyzed the possible mechanism. Methods: We established the arterially perfused rabbit left ventricular wedge preparations to record transmembrane ECG and action potentials of the epicardial cells and endocardial cells simultaneously. We divided two groups by setting the location of premature beats.
本试验以冠状动脉灌注兔左心室楔形组织块标本为研究对象,观察期前刺激位置对单向传导阻滞的时间易损窗的影响、单向传导阻滞和折返激动时间易损窗的关系及折返起源部位等,并分析其可能机制。
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PSS and Ami were administered arterially into denervated kidney in dogs, followed by measuring several renal hemodynamic parameters, including glomerular filtration rate, urine flow, urinary excretion rate of sodium and potassium and etc.
在活体去神经狗肾脏动脉注射PSS和Aml,观察肾小球滤过率、肾血流、钠、钾排泄率等9个肾动力指标。
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In HMI group and AAP10 group, the circumflex coronaries were ligated. In sham group, left thoracotomy was performed without coronary ligation. The animals were feed three months before the study and an arterially perfused rabbit left ventricular wedge preparation was made.
Sham组开胸但不结扎冠状动脉,HMI组和AAP10组开胸并结扎冠状动脉左室支制备心梗模型,普通饲料喂养三个月后制备兔左心室楔形心肌块的灌注模型。
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Transmembrane action potentials were recorded simultaneously from endocardium, epicardium, together with a transmural ECG, in arterially perfused left ventricular preparations by use of 2 separate intracellular floating microelectrodes. The stimulus-response- interval of the epicardium and the incidence of ventricular tachycardia were observed as well.
灌流全程同时采用浮置玻璃微电极法同步记录内膜下心肌、外膜下心肌跨膜动作电位和跨壁ECG,并观察心外膜下心肌的刺激反应间期(Stimulus-response-interval, SRI)和室性心动过速的诱发率。
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