anoxia [æ'nɔksiə]

anoxia的基本解释

n.

缺氧症

与anoxia相关的网络例句 [注：此内容来源于网络，仅供参考]

Besides, HIF-1 can quickly induce a lot of expressions and the adjustment of anoxia-induced genes, so as to enhance the hypoxia-induced genetic transcription and increase the expressions, and engender physiological effects, such as adjusting the transcription of EPO genes to increase their production, thereby the red blood cells quantity is enlarged, which strengthens the ability of oxygen transportation and mitigates anoxia; adjusting VEGF genetic transcription to engender more blood capillaries, consequently the transportation and supply of oxygen and substance of energy source are strengthened; adjusting the glycolytic enzyme to enhance glycolysis ability under anoxemia condition and increase energy supply; functioning on HO-1 genes in vascular smooth muscle cells to produce HO-1, which can engender NO and CO as pneumatic signal molecules to activate the guanylate cyclases, thus CGMP level is improved to make the vascular smooth muscles chalasia, to restrain platelet agglutination, so that theblood flow and vascular permeability are increased, and the anoxic tissues obtain ample oxygen supply, etc..

在低氧环境中，HIF-1可以被很快诱导大量表达并介导低氧诱导基因的调控，使低氧诱导基因转录增强，表达产物增多，产生生理效应。

Methods The model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed. The content of maiondialdehyde,activity of superoxide dismutasein the brain tissues of cerebral ischemia and anoxia mice were measured by TBA,NBT technology respectively.

采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型，观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响；TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

ResultsBVF could remarkably lengthen the survival time of cerebral ischemia and anoxia mice, and the time of catching their breath after mice decollated,could evidently decrease mouse brain capillary vessel permeability and the content of MDA, increase activity of SOD in the brain tissues of cerebral ischemia and anoxia mice.

方法采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型，观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响；TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

与anoxia相关的网络解释 [注：此内容来源于网络，仅供参考]

anoxia：缺氧症

长期的麦角胺作用,能引起组织局部之缺氧症(Anoxia),尤其是末端组织之血液供给缺乏,这些影响再因微血管内皮之伤害,而造成阻塞微血管血流,血管栓塞,停滞和乾涸坏疽.

anoxia：缺氧

子与母体不合(Rh incompatibility)、生产时缺氧(anoxia)窒息或脑损、新瓦登伯革氏症(Waardenberg's syndrome)等. 按照声音传导方式区分,助听器分为空气传导式(air conductive)与头骨传导式(born-anchored)两大类,大部分助听器属空气传导式,

anoxia：缺氧症<在血液和细胞中

anoxemia 缺氧血症 | anoxia 缺氧症<在血液和细胞中 | ANSI = American National Standards Institute 美国国家标准学会

anoxia：无氧症

"anoxemia","血中缺氧" | "anoxia","无氧症" | "antenna","天线"

anemic anoxia：贫血性缺氧

anechoic 无回声的 | anemic anoxia 贫血性缺氧 | anemophobia 恐风症