angina pectoris

angina pectoris的基本解释

n.

心绞痛, 真心痛, 注心痛

与angina pectoris相关的网络例句 [注：此内容来源于网络，仅供参考]

Reaches 1／6 generally in the acute cardiac arrest patient Central Africa typical angina pectoris～1／3.But in the old age acute cardiac arrest case of illness,the nontypical angina pectoris disease incidence rate may be 30.2%,does not have the symptom is reaches as high as 42％,but the typical angina pectoriS only accounts for 20%.

一般在急性心肌梗死患者中非典型心绞痛型达1／6～1／3，而在老年急性心肌梗死病例中，非典型心绞痛型的发病率可为30.2％，无症状型更是高达42％，而典型心绞痛型仅占20％。

Methods By using enzymelinked immunosorbent assay and light scattering rate nephelometry method, levels of sgp130 and hsCRP were measured in 20 patients with acute myocardial infarction, 20 unstable angina pectoris, and 20 stable angina pectoris.

可溶性糖蛋白130（sgp130）和超敏C反应蛋白是反映机体的炎症状态和程度的指标，IL6是冠心病发病中的重要炎性因子，而sgp130则是IL6发挥生物学作用过程中重要的信号分子[2]。

Methods Monocytes in peripheral blood were obtained from 12 patients with acute myocardial infarction, 16 unstable angina pectoris, 12 stable angina pectoris and 8 normals by ficoll density gradient centrifugalization. The cells were incubated for 48 hours to induce them to differentiate into macrophages by adding phorbolester 12-myristate 13-acetate. The levels of MMP-9 and TF in the supernatant were measured by ELISA.

密度梯度离心法提取12名急性心肌梗死、16名不稳定心绞痛、12名稳定型心绞痛患者和8名冠脉正常者的单核细胞，用佛波酯诱导其分化为巨噬细胞，ELISA法测定单核源巨噬细胞上清液MMP-9、TF含量。

与angina pectoris相关的网络解释 [注：此内容来源于网络，仅供参考]

angina pectoris：心绞痛

Heberden在1772年引入了"心绞痛" (angina pectoris)的概念,其特征是一种与活动明显有关的胸部"绞榨和焦虑感(a sense of应当认识到,自从上些能有效地预防冠状动脉疾病进展的措施引入以来,对可能为心源性胸痛病人的诊断方法发了变化.

angina pectoris：狭心症

狭心症(angina pectoris)是冠状动脉供血不足,心肌急速的、暂时缺血与缺氧所导致的临床综合征. 其特点为阵发性的前胸压榨性痛苦悲伤感觉,可伴有其它症状,痛苦悲伤主要位于胸骨后部,可放射至心前区与左上肢,常发生于劳动或者情绪激动时,

angina pectoris：冠心病心绞痛

近年来随着人们生活水平的提高,高血压、糖尿病、高脂血症和吸烟者的增多使冠心病心绞痛(Angina pectoris)的发病率和死亡率有明显的增高,发病年龄也日趋年轻化.

angina pectoris：心绞痛症

angina 咽峡炎 | angina pectoris 心绞痛症 | angina 咽峡炎

unstable angina pectoris：不稳定型心绞痛

近年临床上较为广泛地应用不稳定型心绞痛(unstable angina pectoris)一词,指介于稳定型心绞痛与急性心肌梗塞和猝死之间的临床状态,包括了初发型,恶化型劳累性心绞痛,和各型自发性心绞痛在内,其病理基础是在原有病变上发生冠状动脉内膜下出血,