Results l.Serum concentrations of VCAM-1 in PIH (93.39 + 57.3)ug/ml were higher than that in normal pregnant group. Serum concentrations of VCAM-1 in moderate(97.89 + 34.07)ug/ml and severe PIH(132.24 + 60.97)ug/ml were significantly higher than that in normal group.(P.05, P.01). There was no difference between serum levels of VCAM-1 mild PIH and normal pregnant group. 2.Serum levels of IL-6 in PIH group(102.17 ?48. 31)pg/ml were significantly higher than that in normal group(49.16 + 12.9)pg/ml.Serum levels of IL-6 in moderate(95.79+31.19)pg/ml and severe PIH( 127.27+11.3 8)pg,ml were significantly higher than that in normal group. There was no difference between serum levels of PIH in mild PIH (52.13 + 12.90)pg/ml and normal pregnant group. 3. In PIH group, serum concentrations of VCAM-1 correlate with the levels of IL-6,r=0.63. 4.The expression of VCAM-1 in cytotrophoblast of spiral arteries in normal pregnant group(100%) were significantly higher than that in PIH group.Theexpression of VCAM-1 in moderate PIH(37.50%) and sever PIH were lower than that in normal group.There was no difference between the mild PIH and normal group.Conclusions The increased levels of serum of VCAM-1 may participate in the process of vascular endothelium damages in PIH.

结果 1、妊高征组血清VCAM-1浓度为(93.39±57.3)μg／ml明显高于正常妊娠组(44.87±15.60)μg／ml，差别有显著性(P＜0.05)；中、重度妊高征组VCAM-1浓度分别为(97.89±34.07)μg／ml和(132.24±60.97)μg／ml，与正常妊娠组比较，差异有显著性(p＜0.05)和非常显著性(P＜0.01)；轻度妊高征组VCAH-1为(48.46±15.60)μg／ml与正常妊娠组比较，差异无显著性(P＞0.05)。2、妊高征组血清IL-6含量为(102.17±48.31)pg／ml，明显高于正常妊娠组(49.16+12.9)pg／ml，差异有非常显著性(P＜0.01)；中、重度妊高征组IL-6含量分别(95.79±31.19)pg／ml和(127.27±11.38)pg／ml，与正常妊娠组比较，差异有显著性(P＜0.05)和非常显著性(P＜0.01)；轻度妊高征组IL-6含量为(52.13±12.90)pg／ml与正常妊娠组比较，差异无显著性(P＞0.05)。3、VCAM-1与IL-6水平呈明显正相关，r=0.63(P＜0.01)。4、子宫胎盘床螺旋动脉滋养细胞VCAM-1表达，正常妊娠组都存在阳性表达(阳性表达率100％)，妊高征组有10例阳性表达阳性表达率为叩们，差别有非常显著性河＜0.01L 中、重度妊高征组阳性表达率分别为37、50％和0，与正常妊娠组比较，差别有显著性河＜0.05）和非常显著性汀＜0.01太轻度妊高征组阳性表达率为83.33凡与正常妊娠组差别无显著性问＞0.05）。

Results:Palatopharyngeal data of normal adults and patients with OSAS are as followed,length of softpalate:27.68±7.17 mm,29.94±4.71 mm(P＜0.05);width of uvula:8.40±1.18 mm, 10.74±1.68 mm(p＜0.01);length of uvula:10.20±2.85 mm,11.94±2.84 mm (P＜0.01);distance of pharyx:23.70±7.05 mm,19.87±4.40 mm(P＜0.05);distance of palatopharyx:12.36±3.97 mm,9.80±2.90 mm(P＜0.01).All of which were significantly different.

结果：正常人和OSAS患者所测腭咽解剖指标分别为：软腭长(27.68±7.17)mm，(29.94±4.71)mm(P＜0.05)；悬壅垂宽(8.40±1.18)mm，(10.74±1.68)mm(P＜0.01)；悬雍垂长(10.20±2.85)mm，(11.94±2.48)mm(P＜0.01)；咽侧距(23.70±7.05)mm，(19.87±4.40)mm(P＜0.05)；腭咽距(12.36±3.97)mm，(9.80±2.90)mm(P＜0.01)，均存在显著差异。

There were no significant differences in the normalization of alanine aminotransferase levels between the two groups at week 12 and 24 (52.5% vs 60.0%, 77.5% vs 75.0%). The mean reductions in serum HBV DNA from the baseline levels at week 12 and 24 were similar between the two groups [5.27 vs.5.36, 6.49 vs.6.18 log (on a base-10 scale) copies per milliliter]. More patients in the telbivudine group had HBeAg seroconversion at week12 than those in the entecavir group (20.0% vs 5.0%, P = 0.043); however, there was no significant difference between the two groups at week 24 (27.5% vs 17.5%).

治疗12周时，替比夫定组和恩替卡韦组患者 HBV DNA 低于检测值率均为50.0%， ALT 复常率分别为52.5%和60.0%（ P ＞0.05）， HBeAg 阴转率分别为30.0%和5.0%（ P ＜0.01）， HBeAg 血清学转换率分别为20.0%和5.0%（ P ＜0.05）；在治疗24周时，两组 HBV DNA 低于检测值率分别为80%和70%（ P ＞0.05）， ALT 复常率分别为77.5%和75.0%（ P ＞0.05）， HBeAg 阴转率分别为45.0%和32.5%（ P ＞0.05）， HBeAg 血清学转换率分别为27.5%和17.5%（ P ＞0.05）；两组均未发现明显不良反应。

At this point the P：凤凰在此

25: 一切餐具都洗干净了......Everything was.... 04:18 | 26: 凤凰在此......At this point the P.... 03:14 | 27: 简一下惊醒了......It was Jane who a.... 03:07

unreliable science：不可信的科學

reliable science 可信的科學p.26 | unreliable science 不可信的科學p.27 | core case study 核心專題研究p.28

Gentian Root P.E：秦艽提取物

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