IL

IL的基本解释

abbr.

=Illinois 伊利诺(美国州名)

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[计] 指令表, 中间语言, 解释语言

IL所属的单词分类

City / 城市 [3852]

Alene  ·  Zwijndrecht  ·  Zuchwil  ·  Zonhoven  ·  Zoetermeer  ·  Zittau  ·  Zell am See  ·  Zelandia  ·  Zaventem  ·  Zaragoza

与IL相关的网络例句 [注：此内容来源于网络，仅供参考]

Results The functions of major organs in group HVHF improved obviously,and the MODS morbidity and mortality reduced significantly,compared with that in group HVHF. ENIAF and ECKR combining activity in group HVHF decreased much slowly through the treatment or HVHF than that in group MODS(P 0.01). After hemofiltration the plasma levels of TNF α IL-1β IL-6 IL-8 in group HVHF descended significantly compared with those in group MODS (P.01).Except TNF α, IL-1β.IL-6 IL-8 and IL-10 were detectable in ultrafiltrate.

结果 与MODS组相比，HVHF组各主要脏器功能指标明显改善，MODS的发生率和死亡率显著降低，滤过治疗后ENIAF和ECKR结合活性下降幅度明显趋缓；HVHF组各促炎因子血浆水平明显下降，与MODS组相比有显著差异(P.01)；除TNFα外，IL-1β、IL-6、IL-8、IL-10均在超滤液中检测到。

Results: 1 The capacity of engulfment of A549 cells started enhancing at the 1h of preincubation with dexamethasone, reached the highest at the 4h of preincubation,At the 8h、24h of preincubation the capacity of engulfment evidently reduced. 2 Glucocorticoid cytosolic receptors antagonist RU38486 inhibited the improved engulfment ability by dexamethasone. 3Phagocytosing apoptotic eosinophils did not change the release of IL-6 and IL-8,but can inhibited the release of IL-6 and IL-8 from A549 cells stimulated by lipopolysaccharide. 4Increasely uptaking by dexamethasone also did not agitate the release of IL-6 and IL-8 from A549 cells. Conclusion: 1Dexamethasome improve the phagocyte capacity for apoptotic eosinophils of A549 cells by glucocorticoid cytosolic receptors, and the effect presents time-dependently. 2Phagocytosis of apoptotic eosinphils can degrade inflammatory reflection, so it is likely to be critical in the control of asthma inflammation.

结果： 1 A549细胞在地塞米松诱导后1小时吞噬能力开始升高，4小时吞噬能力达最高峰，8小时、24小时吞噬能力明显下降 2加入RU38486后抑制了地塞米松所增强的吞噬能力 3凋亡嗜酸粒细胞的吞噬并不能改变A549细胞释放IL-6和IL-8，但能抑制脂多糖刺激A549细胞后IL-6、IL-8的分泌 4地塞米松增加的吞噬也不激发A549细胞释放IL-6和IL-8 结论： 1地塞米松促进A549细胞吞噬凋亡的嗜酸粒细胞是通过A549细胞糖皮质激素胞浆受体发挥作用的，并且具有时间依赖性 2 A549细胞吞噬凋亡的嗜酸粒细胞能降低炎症反应，对哮喘炎症的控制具有重要意义

Conbining previous results with our results, we conclude that the soluble gp130 mutant containing CBD can antagonize the biological activities of IL-6 by competitively binding IL-6/IL-6Rα with wild-type gp130, and the membraneanchored mutant could transduce IL-6 signal, but its efficiancy was much lower than that of wild-type gp130, therefore, we thought the Ig-like module also played an important role in IL-6 signal transduction by involving in the formation of a hexameric IL-6 receptor complex.

结合以前的研究结果，我们认为只含胞外CBD区的gp130突变体通过与膜结合型gp130竟争结合IL-6／IL-6Rα复合物而具有拮抗IL-6功能的作用；而只含胞外CBD区的gp130突变体，尽管仍能传导IL-6信号，但其效率远低于野生型gp130，因此，我们认为gp130的Ig样区在IL-6转导中也起着十分重要的作用。

与IL相关的网络解释 [注：此内容来源于网络，仅供参考]

il : Israel：以色列

ie : Ireland , 爱尔兰共和国 | il : Israel , 以色列 | in : India , 印度

il : Israel：以色列 亚洲

CY Cyprus 塞浦路斯 亚洲 | IL Israel 以色列 亚洲 | IN India 印度 亚洲

il : Israel：以色列(域名)

IICS IsotopesInformationCenter 同位素信息中心(美国) | IL Israel 以色列(域名) | IWM IssuerWorkingKey 发行商的工作密钥

il : Israel：以色列 LGG 比利时列日 中东

84 IE IRELAND 爱尔兰 LGG 比利时列日 欧 | 85 IL ISRAEL 以色列 LGG 比利时列日 中东 | 86 IT ITALY 意大利 LGG 比利时列日 欧

.il：以色列

[地址]以色列赫兹利亚皮图亚奇 [申请人]塔罗制药工业有限公司 [公开号]1571661 [国家省市]以色列(IL) [国际分类]A61K9/16、A61K9/50 [摘要] 一种用于口服的抗溢出药物制剂,包括药剂和抗溢出成分,所述药剂和成分在没有遮味剂存在的情况下有苦味,