ASE

ASE的基本解释

n.

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abbr.

American Stock Exchange 美国证券交易所

与ASE相关的网络例句 [注：此内容来源于网络，仅供参考]

A flat high efficiency super-broadband source was presented. We achieved a C+L-band superfluorescent source, in the region from 1524. 0 to 1600.4nm, the power of the ASE are over -12. 0dBm and the ASE profile excursion is only ±1. 4dB in the region from 1539. 2 to 1600.4nm. The power of the ASE is 22. 1mW and the power conversion efficiency is 18. 8%. 9. The amplification characteristics of thulium-doped fiber amplifier had been studied using the basic theory for fiber amplifier. The connection between the Tm-doped fluoride fiber and silicon fiber was accomplished, and we studied the TDFA and the reason that the expecting results hadn't achieved had been analyzed. The assumption of the Er-Tm-co-doped silicon fiber had been presented, and we studied the Er-Tm-co-doped silicon in cooperation with the correlative corporation, some results had been got and the further studies are carried on.

实验结果得到了覆盖C+L-波段的超荧光光源，在1524.0-1600.4nm波长范围内，自发辐射谱的功率均高于-12.0dBm；在1539.2-1600.4nm波长范围内，自发辐射谱不平坦度为±1.4dB；荧光总功率为22.1mW，功率转换效率为18.8%； 9、应用光纤放大器的基本理论，理论分析和研究了掺铥光纤放大器的放大特性；解决了氟化物掺铥光纤与硅基光纤的连接问题，进行了TDFA的实验研究，分析了没有得到预期结果的原因；最后提出了铒-铥共掺硅基光纤的构想，并与有关单位合作，共同研制了铒-铥共掺硅基光纤，得到了一些前期结果，进一步的研究仍在进行当中。

ATP is formed in the payoff phase of glycolysis by two reactions, promoted by phosphoglycerate k in ase and pyruvate k in ase.

ATP在糖酵解的收获阶段经过两个反应生成，由磷酸甘油酸激酶和丙酮酸激酶催化。

TyPe II collagen induced arthritisln the rat ank1e joint andoVathumin as antigen induced arthritis WA in the rabbit knee joint wereestab1ish2 Qualitative evaluation of me in skin, muscle, synovium, cedilagearound joint and blood was performed by OMA3 The CIA rats were treated on day 7 after hind paw swelling and erythemaAnimals were injected intravenously with ase at a dose of 10mg/kg,tWenty minuots 1ater, one ankle of the rats random1y assigned was exPosedlaser irradiation at l00J/cm fOr l000 seconds, and another ankle wasM grouP wihout laser The other two groups is unmanipulatedcontrol group and untreated CIA group Bimaleolar ankle widthmeasuremellts were taken in all animals every tWo days using amicrometer The histopathology of the ank1e Joint was assessed at day 21after disease onset4 The pro1iferating cell nuclear antigen WCNA of CIA treated by PDT andthe HMME group without laser was doterdrined by immunohistochemiStry5 The AfA rabbits were treated on day 7 after knee swelling and erythemaThe theraPy invo1ved lntravenous injection of l0mg/kg HMME, fOl1owedby 20 minues period in dim light, and transdermal light treatment with\l00 J/cm2 fOr l000 seconds The inner sides of the treated Anees wereirradiated at first, and then the outer side did 24 hours later, the synovialtissue of the Anees joint were removed and in situ cel1 aPoptosis wasdetCCted With tednal deoxync1eotidyl transferase-mediated dUTP nickend labelingR6suIt8:l The pathologic changes of CIA and AIA include subsynovial inflammation,opovial hyPerplasia, pannus formation, cartilage and bone destructionresemble RA.2 The studies demonstrated that there are different uptake of HMME withinskin, muscle, synovium, cartilage and b1ood, and the synovium cou1draPidly uPtake more ase than skin and cartilage at the firSt 30 minuesaller intravenous injection of HMME3 The bimaleolar anke width had no different among PDT treated group,H group withollt 1aser and untreated CIA group But hlstologicalevaluation showed statiStical1y significallt reductions in synovialhyperplasia, pannus formation and cart1lage reosion, bone destruction andtotal score in PDT treated group4 Image analysis showed that the ratlo bforeen the areas of the coufltedobect to that of the entire area in PDTtreated grOup is lower than that in conirol group, but the integrated oPticaldensity had no different between the two groups5 Imape analysis showed that the ratio between the area of the countedobject to that of the e

治疗组在大鼠出现踝关节红肿后1周，炎症达到高峰时进行PDT治疗。随机治疗大鼠一侧的踝关节，另。2。一一侧作单纯HMME 对照。治疗方法是大鼠麻醉后尾静脉注入 HMME10ngkg，20分钟后踝关节照光，激光波长627.sum，功率密度 100mwcm'，照射时间1000秒，能量密度100）／。治疗后避光喂养72 小时。隔日一次测量大鼠的踝关节左右横径，治疗后两周取关节进行病理d 观察。 4。大鼠CIA模型用上述方法进行PDT治疗后，治疗组和单纯HMME 组用兔疫组化SP法检测石蜡切片的核增殖抗原。 5。兔AIA模型在关节炎出现第七天进行PDT治疗，随机治疗一侧膝关节，另一侧作自身对照。兔耳静脉注入I'arrainrelomg／Kg，20分钟后，膝关节用金蒸气激光照射，激光能量密度100）儿旷。24 ／J'时后取膝关节滑膜作病理检查，并用脱氧核昔酸末端转移酶介导的缺口末端标记法原位检测凋亡细胞。结果： 1。模型观察：CIA大鼠炎症高峰期滑膜下炎细胞浸润明显，滑膜细胞明显增殖，炎症达到高峰后二周，血管缀形成，并侵蚀和破坏软骨和骨， CIA模型病理改变与人类RA相似。兔AIA模型膝关节滑膜病理可见滑膜细胞增生，滑膜下炎细胞浸润，也与人类RA滑膜改变相似。 2。关节周围组织中光敏剂含量的测定结果表明，各组织对HMME 的吸收速度和吸收量不同，荧光值一时间曲线不同，滑膜组织比皮肤和软骨对 HMME的吸收多，在 2 0分钟时即有明显差异。 3.PDT对CIA模型的治疗结果表明：PDT治疗后关节炎组、单纯 HMME组和治疗组踝关节左右横径统计学检验差异没有显著性，但病理评分PDT治疗组滑膜增生、血管资形成及软骨破坏、骨破坏和总分比关节炎对照组和HMME对照组好，统计学检验差异有显著性。。3＿军医进修学院硕士学位论文中文摘要 4.PDT治疗组PCNA阳性细胞较对照组少，图像分析结果表明面密度（阳性染色的面积总和与统计视野面积的比值）治疗组小于对照组，统计学检验差异有显著性。。 5.PDT治疗组凋亡阳性细胞较对照组明显增多，图像分析结果单位视野内阳性细胞数和面密度PDT治疗组高于对照组，统计学检验差异有显著性。凋亡细胞核直径PDT治疗组较小，与对照组相比，统计学检验差异有显著性。结论：二。CIA、AIA的病理改变类似人类RA，可作为研究RA病因、发病机制、检查及治疗方法的模型。 2。各组织对HMME的吸收速度和吸收量不同，滑膜组织比皮。

与ASE相关的网络解释 [注：此内容来源于网络，仅供参考]

aseexport ASE：输出 ASE 汇出

aseadmin ASE 管理 ASE 管理 | aseexport ASE 输出 ASE 汇出 | aselinks ASE 链接 ASE 连结

aseexport ASE：输入 ASE 汇出

aseadmin ASE 管理 ASE 管理 | aseexport ASE 输入 ASE 汇出 | aselinks ASE 链接 ASE 连结

aserows ASE：行 ASE 列

aselinks ASE 链接 ASE 连结 | aserows ASE 行 ASE 列 | aseselect ASE 选择 ASE 选取